Neuroinflammatory processes are augmented in mice overexpressing human heat-shock protein B1 following ethanol-induced brain injury

Neuroinflammatory processes are augmented in mice overexpressing human heat-shock protein B1 following ethanol-induced brain injury

Abstract Background Heat-shock protein B1 (HSPB1) is among the most well-known and versatile member of the evolutionarily conserved family of small heat-shock proteins. It has been implicated to serve a neuroprotective role against various neurological disorders via its modulatory activity on inflam...

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Bibliographic Details
Main Authors: Brigitta Dukay, Fruzsina R. Walter, Judit P. Vigh, Beáta Barabási, Petra Hajdu, Tamás Balassa, Ede Migh, András Kincses, Zsófia Hoyk, Titanilla Szögi, Emőke Borbély, Bálint Csoboz, Péter Horváth, Lívia Fülöp, Botond Penke, László Vígh, Mária A. Deli, Miklós Sántha, Melinda E. Tóth
Format: Article
Language:English
Published: BMC 2021-01-01
Series:Journal of Neuroinflammation
Subjects:
hHSPB1
Heat-shock protein
Astroglia
Cytokines
Ethanol toxicity
Microglia
Online Access:https://doi.org/10.1186/s12974-020-02070-2

Internet

https://doi.org/10.1186/s12974-020-02070-2

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