Arginine transport and other determinants of nitric oxide production in human septic shock
The arterial vasodilation seen in human septic shock is conventionally attributed to increased nitric oxide (NO) production, primarily by extrapolation of animal and cellular studies. Little is known of the cellular source of NO in human septic shock. Other mediators, such as carbon monoxide (CO), m...
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University of Oxford
2002
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Online Access: | http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275376 |