Investigation of mesalazine as an antifibrotic drug following myocardial infarction in male mice

Abstract Objectives Myocardial infarction (MI) initiates a complex reparative response during which damaged cardiac muscle is replaced by connective tissue. While the initial repair is essential for survival, excessive fibrosis post‐MI is a primary contributor to progressive cardiac dysfunction, and...

Full description

Bibliographic Details

Published in:	Physiological Reports
Main Authors:	Stephan R. Künzel, Luise Winter, Maximilian Hoffmann, Theresa A. Kant, Jessica Thiel, Romy Kronstein‐Wiedemann, Erik Klapproth, Kristina Lorenz, Ali El‐Armouche, Susanne Kämmerer
Format:	Article
Language:	English
Published:	Wiley 2023-09-01
Subjects:	drug repurposing fibrosis mesalazine myocardial infarction
Online Access:	https://doi.org/10.14814/phy2.15809