The Improvement of Experimentally Induced Gastric Ulcers in Rats by Inhibiting Vascularization Through the Blocking of the TNF‐α Type 1 Receptor

ABSTRACT Background About 5%–10% of the world's population is affected by gastric ulcers, which can result in gastrointestinal perforation and bleeding. Consequently, we aimed to investigate whether blocking TNF‐α type 1 receptor (TNFR1) with CAY10500 could diminish experimentally induced gastr...

Full description

Bibliographic Details

Published in:	Immunity, Inflammation and Disease
Main Authors:	Abdullah Alattar, Reem Alshaman, Fawaz E. Alanazi, Omar Bahattab, Hanan M. Hassan, Mohammed M. H. Al‐Gayyar
Format:	Article
Language:	English
Published:	Wiley 2025-10-01
Subjects:	extracellular signal‐regulated kinase (ERK) gastric ulcer (GU) intercellular adhesion molecule (ICAM)‐1 phosphatidylinositol‐3 kinase (PI3K) protein kinase B (PKB, AKT) tumor necrosis factor‐α type 1 receptor (TNFR1)
Online Access:	https://doi.org/10.1002/iid3.70279