Ferroptosis and inflammation are modulated by the NFIL3-ACSL4 axis in sepsis associated-acute kidney injury

Abstract Sepsis-associated acute kidney injury (SA-AKI) increases the risk of death in patients with sepsis, and its major pathological change is the death of renal tubular cells. However, the mechanism of its occurrence remains unclear. Sepsis can lead to circadian dysregulation, and the rhythm gen...

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Bibliographic Details

Published in:	Cell Death Discovery
Main Authors:	Zhong Xiao, Jie Zhang, Zhimin Qiu, Hongbing Liu, Hua Ding, Hi Li, Yuanxin Liu, Xiaohua Zou, Juan Long
Format:	Article
Language:	English
Published:	Nature Publishing Group 2024-08-01
Online Access:	https://doi.org/10.1038/s41420-024-02113-0