Neurorehabilitation in stroke produced by vertebral artery dissection: case presentation
Arterial dissections are a common cause of stroke in the young (mean age 44 to 46 years). Primary lesion is a tear of the arterial intima, which promotes platelet aggregation, thrombus formation, which further produced vessel stenosis / occlusion, distal embolism or vessel wall rupture. Vertebral ar...
Main Authors: | , , , |
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Format: | Article |
Language: | English |
Published: |
Romanian Association of Balneology, Editura Balneara
2018-02-01
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Series: | Balneo Research Journal |
Subjects: | |
Online Access: | http://bioclima.ro/Balneo168.pdf |
Summary: | Arterial dissections are a common cause of stroke in the young (mean age 44 to 46 years). Primary lesion is a tear of the arterial intima, which promotes platelet aggregation, thrombus formation, which further produced vessel stenosis / occlusion, distal embolism or vessel wall rupture. Vertebral artery (VA) dissection appear most commonly in extracranial segments V2 and V3, and could be spontaneous (with underlying predispositions) or triggered by various traumatisms. Clinicaly, VA dissection produces an ischemic stroke or transient ischemic attack , preceded by local symptoms such as neck pain or headache. The diagnosis is confirmed by neurovascular imaging. Treatment of symptomatic VA dissections respect indications of treatment in ischemic strokes. Prognosis is mostly favorable in extracranial dissections.
We present the case of a left VA dissection in V2 segment, produced by physical effort (swimming), which causes 2 ischemic lesions, one in the territory of the left posterior cerebral artery and the other in the territory of the left posterolateral chorroidal artery. Patient’s treatment included antiplatelet agents, statines, and an adapted physical rehabilitation program. At three months he showed significant clinical improvement with regain of autonomy and partial recanalisation at angio-MRI of the V2 segment of the dissected artery. |
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ISSN: | 2069-7597 2069-7619 |