Neurorehabilitation in stroke produced by vertebral artery dissection: case presentation
Arterial dissections are a common cause of stroke in the young (mean age 44 to 46 years). Primary lesion is a tear of the arterial intima, which promotes platelet aggregation, thrombus formation, which further produced vessel stenosis / occlusion, distal embolism or vessel wall rupture. Vertebral ar...
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doaj-0067b2f2331542e380c52dae57c2d5b52020-11-25T00:23:20ZengRomanian Association of Balneology, Editura BalnearaBalneo Research Journal2069-75972069-76192018-02-0191343710.12680/balneo.2018.168Neurorehabilitation in stroke produced by vertebral artery dissection: case presentationStanescu Ioana0Bulboaca Adriana1Kallo Rita2Dogaru Gabriela3Department of Neurosciences, University of Medicine and Pharmacy Cluj-Napoca, RomaniaDepartment of Physiopathology, University of Medicine and Pharmacy Cluj-Napoca, RomaniaClinical Rehabilitation Hospital Cluj-Napoca, RomaniaDepartment of Medical Rehabilitation University of Medicine and Pharmacy Cluj-Napoca, RomaniaArterial dissections are a common cause of stroke in the young (mean age 44 to 46 years). Primary lesion is a tear of the arterial intima, which promotes platelet aggregation, thrombus formation, which further produced vessel stenosis / occlusion, distal embolism or vessel wall rupture. Vertebral artery (VA) dissection appear most commonly in extracranial segments V2 and V3, and could be spontaneous (with underlying predispositions) or triggered by various traumatisms. Clinicaly, VA dissection produces an ischemic stroke or transient ischemic attack , preceded by local symptoms such as neck pain or headache. The diagnosis is confirmed by neurovascular imaging. Treatment of symptomatic VA dissections respect indications of treatment in ischemic strokes. Prognosis is mostly favorable in extracranial dissections. We present the case of a left VA dissection in V2 segment, produced by physical effort (swimming), which causes 2 ischemic lesions, one in the territory of the left posterior cerebral artery and the other in the territory of the left posterolateral chorroidal artery. Patient’s treatment included antiplatelet agents, statines, and an adapted physical rehabilitation program. At three months he showed significant clinical improvement with regain of autonomy and partial recanalisation at angio-MRI of the V2 segment of the dissected artery.http://bioclima.ro/Balneo168.pdfvertebral arteryextracranial dissectioncervical arteriesischemic strokerehabilitation |
collection |
DOAJ |
language |
English |
format |
Article |
sources |
DOAJ |
author |
Stanescu Ioana Bulboaca Adriana Kallo Rita Dogaru Gabriela |
spellingShingle |
Stanescu Ioana Bulboaca Adriana Kallo Rita Dogaru Gabriela Neurorehabilitation in stroke produced by vertebral artery dissection: case presentation Balneo Research Journal vertebral artery extracranial dissection cervical arteries ischemic stroke rehabilitation |
author_facet |
Stanescu Ioana Bulboaca Adriana Kallo Rita Dogaru Gabriela |
author_sort |
Stanescu Ioana |
title |
Neurorehabilitation in stroke produced by vertebral artery dissection: case presentation |
title_short |
Neurorehabilitation in stroke produced by vertebral artery dissection: case presentation |
title_full |
Neurorehabilitation in stroke produced by vertebral artery dissection: case presentation |
title_fullStr |
Neurorehabilitation in stroke produced by vertebral artery dissection: case presentation |
title_full_unstemmed |
Neurorehabilitation in stroke produced by vertebral artery dissection: case presentation |
title_sort |
neurorehabilitation in stroke produced by vertebral artery dissection: case presentation |
publisher |
Romanian Association of Balneology, Editura Balneara |
series |
Balneo Research Journal |
issn |
2069-7597 2069-7619 |
publishDate |
2018-02-01 |
description |
Arterial dissections are a common cause of stroke in the young (mean age 44 to 46 years). Primary lesion is a tear of the arterial intima, which promotes platelet aggregation, thrombus formation, which further produced vessel stenosis / occlusion, distal embolism or vessel wall rupture. Vertebral artery (VA) dissection appear most commonly in extracranial segments V2 and V3, and could be spontaneous (with underlying predispositions) or triggered by various traumatisms. Clinicaly, VA dissection produces an ischemic stroke or transient ischemic attack , preceded by local symptoms such as neck pain or headache. The diagnosis is confirmed by neurovascular imaging. Treatment of symptomatic VA dissections respect indications of treatment in ischemic strokes. Prognosis is mostly favorable in extracranial dissections.
We present the case of a left VA dissection in V2 segment, produced by physical effort (swimming), which causes 2 ischemic lesions, one in the territory of the left posterior cerebral artery and the other in the territory of the left posterolateral chorroidal artery. Patient’s treatment included antiplatelet agents, statines, and an adapted physical rehabilitation program. At three months he showed significant clinical improvement with regain of autonomy and partial recanalisation at angio-MRI of the V2 segment of the dissected artery. |
topic |
vertebral artery extracranial dissection cervical arteries ischemic stroke rehabilitation |
url |
http://bioclima.ro/Balneo168.pdf |
work_keys_str_mv |
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1725357582279770112 |