Modified cytoplasmic Ca2+ sequestration contributes to spinal cord injury-induced augmentation of nerve-evoked contractions in the rat tail artery.
In rat tail artery (RTA), spinal cord injury (SCI) increases nerve-evoked contractions and the contribution of L-type Ca2+ channels to these responses. In RTAs from unoperated rats, these channels play a minor role in contractions and Bay K8644 (L-type channel agonist) mimics the effects of SCI. Her...
Main Authors: | Hussain Al Dera, Brid P Callaghan, James A Brock |
---|---|
Format: | Article |
Language: | English |
Published: |
Public Library of Science (PLoS)
2014-01-01
|
Series: | PLoS ONE |
Online Access: | http://europepmc.org/articles/PMC4211886?pdf=render |
Similar Items
-
Spinal cord injury increases the reactivity of rat tail artery to angiotensin II
by: Hussain eAl Dera, et al.
Published: (2015-01-01) -
Mechanomyography and Torque during FES-Evoked Muscle Contractions to Fatigue in Individuals with Spinal Cord Injury
by: Nor Zainah Mohamad, et al.
Published: (2017-07-01) -
Peripheral nerve injury increases glutamate-evoked calcium mobilization in adult spinal cord neurons
by: Doolen Suzanne, et al.
Published: (2012-07-01) -
Repetitive magnetic stimulation affects the microenvironment of nerve regeneration and evoked potentials after spinal cord injury
by: Jin-lan Jiang, et al.
Published: (2016-01-01) -
Nerve injury evoked loss of latexin expression in spinal cord neurons contributes to the development of neuropathic pain.
by: Hilmar Nils Kühlein, et al.
Published: (2011-04-01)