Hyperkalemia on ECG

History of present illness: A 34-year-old diabetic female presented to the emergency department with chest pain status-post AICD firing. She described the pain as a “12 out of 10” which woke her from sleep at 0200, one hour prior to arrival. Vitals were unremarkable. She had no known history of ren...

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Main Author: Bryson Hicks
Format: Article
Language:English
Published: eScholarship Publishing, University of California 2016-09-01
Series:Journal of Education and Teaching in Emergency Medicine
Subjects:
ECG
Online Access:http://jetem.org/hyperkalemia/
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spelling doaj-15c7fc9a25cc464aadffdb172d271be92020-11-24T23:39:35ZengeScholarship Publishing, University of CaliforniaJournal of Education and Teaching in Emergency Medicine2474-19492474-19492016-09-0112V7V8doi:10.21980/J8K017Hyperkalemia on ECGBryson Hicks0University of California, IrvineHistory of present illness: A 34-year-old diabetic female presented to the emergency department with chest pain status-post AICD firing. She described the pain as a “12 out of 10” which woke her from sleep at 0200, one hour prior to arrival. Vitals were unremarkable. She had no known history of renal failure. Due to frequent ED visits for chronic pain, patient had difficult vascular access and nursing was initially unable to obtain IV access. An abnormal rhythm was noted on the cardiac monitor, and ECG was ordered. Significant findings: Initial ECG shows tall, peaked T waves, most prominently in V3 and V4, as well as QRS widening. These findings are consistent with hyperkalemia, which was promptly treated. Follow-up ECG post-treatment shows narrowing of the QRS complexes and normalization of peaked T waves. Discussion: The etiology of hyperkalemia may be due to an acute insult such as crush injury, drug side effect, or in acute renal failure, but may also occur in the setting of a chronic insult such as chronic kidney disease.1 As potassium rises, several abnormalities can be identified on ECG. Initially the T waves become peaked and the QRS complexes widen.2,3 This can devolve into a wide complex rhythm, ventricular tachycardia, ventricular fibrillation, or asystole. Patients may also experience systemic symptoms such as weakness or paralysis.1 In this particular case, labs showed a potassium of 7.6-mmol/L after initial treatment (see repeat EKG). While the incidence of hyperkalemia in the general population is not defined, the incidence in hospitalized patients is 1.3-10%.4-8 Impaired kidney function is the most common risk factor found in 33-83% of affected patients.4,5,8,9 Treatment for hyperkalemia generally includes IV insulin and IV dextrose and nebulized albuterol for intracellular shift of potassium, IV furosemide and IV fluids for dilution and renal excretion of furosemide, and IV calcium for stabilization of cardiac membranes.2,3http://jetem.org/hyperkalemia/CardiologyhyperkalemiaelectrocardiogramECGpeaked T waveswidened QRS
collection DOAJ
language English
format Article
sources DOAJ
author Bryson Hicks
spellingShingle Bryson Hicks
Hyperkalemia on ECG
Journal of Education and Teaching in Emergency Medicine
Cardiology
hyperkalemia
electrocardiogram
ECG
peaked T waves
widened QRS
author_facet Bryson Hicks
author_sort Bryson Hicks
title Hyperkalemia on ECG
title_short Hyperkalemia on ECG
title_full Hyperkalemia on ECG
title_fullStr Hyperkalemia on ECG
title_full_unstemmed Hyperkalemia on ECG
title_sort hyperkalemia on ecg
publisher eScholarship Publishing, University of California
series Journal of Education and Teaching in Emergency Medicine
issn 2474-1949
2474-1949
publishDate 2016-09-01
description History of present illness: A 34-year-old diabetic female presented to the emergency department with chest pain status-post AICD firing. She described the pain as a “12 out of 10” which woke her from sleep at 0200, one hour prior to arrival. Vitals were unremarkable. She had no known history of renal failure. Due to frequent ED visits for chronic pain, patient had difficult vascular access and nursing was initially unable to obtain IV access. An abnormal rhythm was noted on the cardiac monitor, and ECG was ordered. Significant findings: Initial ECG shows tall, peaked T waves, most prominently in V3 and V4, as well as QRS widening. These findings are consistent with hyperkalemia, which was promptly treated. Follow-up ECG post-treatment shows narrowing of the QRS complexes and normalization of peaked T waves. Discussion: The etiology of hyperkalemia may be due to an acute insult such as crush injury, drug side effect, or in acute renal failure, but may also occur in the setting of a chronic insult such as chronic kidney disease.1 As potassium rises, several abnormalities can be identified on ECG. Initially the T waves become peaked and the QRS complexes widen.2,3 This can devolve into a wide complex rhythm, ventricular tachycardia, ventricular fibrillation, or asystole. Patients may also experience systemic symptoms such as weakness or paralysis.1 In this particular case, labs showed a potassium of 7.6-mmol/L after initial treatment (see repeat EKG). While the incidence of hyperkalemia in the general population is not defined, the incidence in hospitalized patients is 1.3-10%.4-8 Impaired kidney function is the most common risk factor found in 33-83% of affected patients.4,5,8,9 Treatment for hyperkalemia generally includes IV insulin and IV dextrose and nebulized albuterol for intracellular shift of potassium, IV furosemide and IV fluids for dilution and renal excretion of furosemide, and IV calcium for stabilization of cardiac membranes.2,3
topic Cardiology
hyperkalemia
electrocardiogram
ECG
peaked T waves
widened QRS
url http://jetem.org/hyperkalemia/
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