Cerebrospinal fluid xanthochromia after pegasparaginase hepatotoxicity in B‐cell acute lymphoblastic leukemia
Key Clinical Message Patients with B‐lineage acute lymphoblastic leukemia treated with pegasparaginase‐containing regimens can develop hepatotoxicity related to it. The systemic hyperbilirubinemia due to hepatotoxicity can lead to the development of CSF xanthochromia.
Main Authors: | Riccardo Moia, Mariangela Greco, Paola Boggione, Maura Nicolosi, Riccardo Bruna, Andrea Patriarca, Gianluca Gaidano, Monia Lunghi |
---|---|
Format: | Article |
Language: | English |
Published: |
Wiley
2020-11-01
|
Series: | Clinical Case Reports |
Subjects: | |
Online Access: | https://doi.org/10.1002/ccr3.3114 |
Similar Items
-
Can CSF spectrophotometry for “Xanthochromia” be used to detect leaking subarachnoid aneurysms in patients with sickle cell anemia with negative MRI or CT angiogram despite hyperbilirubinemia?
by: Wan Yung Siu, et al.
Published: (2020-05-01) -
Safety, efficacy, and clinical utility of asparaginase in the treatment of adult patients with acute lymphoblastic leukemia
by: Koprivnikar J, et al.
Published: (2017-03-01) -
First-line treatment of acute lymphoblastic leukemia with pegasparaginase
by: Riccardo Masetti, et al.
Published: (2009-07-01) -
Nonne-Froin Sign– Dilemma for Subarachnoid Block
by: Vijay Adabala, et al.
Published: (2019-04-01) -
Pseudo-Froin's syndrome, xanthochromia with high protein level of cerebrospinal fluid
by: Soon-Kul Kwon, et al.
Published: (2014-12-01)