Drug-induced tardive dyskinesia
Tardive dyskinesia (TD) is a type caused by the use of different medications. The pathogenesis of TD is associated with dopamine receptor blockade, gamma-aminobutyric acid depletion, cholinergic deficiency, oxidative stress, impaired synaptic plasticity, and neurotoxicity. TD most often occurs after...
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doaj-33b91fe3e4a942f385f7106e83fd9b0f2021-07-29T08:58:41ZrusIMA-PRESS LLCNevrologiâ, Nejropsihiatriâ, Psihosomatika2074-27112310-13422020-02-01121818610.14412/2074-2711-2020-1-81-86959Drug-induced tardive dyskinesiaT. M. Ostroumova0V. A. Tolmacheva1O. D. Ostroumova2V. A. Parfenov3Department of Nervous System Diseases and Neurosurgery, Institute of Clinical Medicine, I.M. Sechenov First Moscow State Medical University (Sechenov University), Ministry of Health of RussiaDepartment of Nervous System Diseases and Neurosurgery, Institute of Clinical Medicine, I.M. Sechenov First Moscow State Medical University (Sechenov University), Ministry of Health of RussiaRussian Medical Academy of Continuing Medical Education, Ministry of Health of Russia; N.I. Pirogov Russian National Research Medical University, Ministry of Health of RussiaDepartment of Nervous System Diseases and Neurosurgery, Institute of Clinical Medicine, I.M. Sechenov First Moscow State Medical University (Sechenov University), Ministry of Health of RussiaTardive dyskinesia (TD) is a type caused by the use of different medications. The pathogenesis of TD is associated with dopamine receptor blockade, gamma-aminobutyric acid depletion, cholinergic deficiency, oxidative stress, impaired synaptic plasticity, and neurotoxicity. TD most often occurs after taking metoclopramide, antipsychotics, antidepressants, calcium channel blockers, and other drugs.The review considers an adequate assessment of the potential risk of TD when the patient uses prescribed drugs, as well as the monitoring and correction of risk factors for TD, discontinuation or replacement of drugs that potentially cause TD, as well as its treatment.https://nnp.ima-press.net/nnp/article/view/1267tardive dyskinesiaadverse reactionsdrug-induced dyskinesia |
collection |
DOAJ |
language |
Russian |
format |
Article |
sources |
DOAJ |
author |
T. M. Ostroumova V. A. Tolmacheva O. D. Ostroumova V. A. Parfenov |
spellingShingle |
T. M. Ostroumova V. A. Tolmacheva O. D. Ostroumova V. A. Parfenov Drug-induced tardive dyskinesia Nevrologiâ, Nejropsihiatriâ, Psihosomatika tardive dyskinesia adverse reactions drug-induced dyskinesia |
author_facet |
T. M. Ostroumova V. A. Tolmacheva O. D. Ostroumova V. A. Parfenov |
author_sort |
T. M. Ostroumova |
title |
Drug-induced tardive dyskinesia |
title_short |
Drug-induced tardive dyskinesia |
title_full |
Drug-induced tardive dyskinesia |
title_fullStr |
Drug-induced tardive dyskinesia |
title_full_unstemmed |
Drug-induced tardive dyskinesia |
title_sort |
drug-induced tardive dyskinesia |
publisher |
IMA-PRESS LLC |
series |
Nevrologiâ, Nejropsihiatriâ, Psihosomatika |
issn |
2074-2711 2310-1342 |
publishDate |
2020-02-01 |
description |
Tardive dyskinesia (TD) is a type caused by the use of different medications. The pathogenesis of TD is associated with dopamine receptor blockade, gamma-aminobutyric acid depletion, cholinergic deficiency, oxidative stress, impaired synaptic plasticity, and neurotoxicity. TD most often occurs after taking metoclopramide, antipsychotics, antidepressants, calcium channel blockers, and other drugs.The review considers an adequate assessment of the potential risk of TD when the patient uses prescribed drugs, as well as the monitoring and correction of risk factors for TD, discontinuation or replacement of drugs that potentially cause TD, as well as its treatment. |
topic |
tardive dyskinesia adverse reactions drug-induced dyskinesia |
url |
https://nnp.ima-press.net/nnp/article/view/1267 |
work_keys_str_mv |
AT tmostroumova druginducedtardivedyskinesia AT vatolmacheva druginducedtardivedyskinesia AT odostroumova druginducedtardivedyskinesia AT vaparfenov druginducedtardivedyskinesia |
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1721250486739271680 |