Data on the evaluation of FGF2 gene expression in Colorectal Cancer

Data on the evaluation of FGF2 gene expression in Colorectal Cancer

The data presented in this article is related with the research paper entitled “Evaluation of MGP gene expression in colorectal cancer”, available on Gene journal [1]. From all the transcription factors known to regulate MGP, FGF2 is the most described in colon adenocarcinoma and colon tumor cell li...

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Main Authors: Helena Caiado, Natércia Conceição, Daniel Tiago, Ana Marreiros, Susana Vicente, Jose Luis Enriquez, Ana Margarida Vaz, Artur Antunes, Horácio Guerreiro, Paulo Caldeira, M. Leonor Cancela
Format: Article
Language:English
Published: Elsevier 2020-08-01
Series:Data in Brief
Subjects:
Colorectal Cancer (CRC)
Matrix gla protein
Gene expression
Transcription factors
FGF2
Online Access:http://www.sciencedirect.com/science/article/pii/S2352340920306594
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spelling doaj-37f7dd72c87b4fa09f0dafcb999b46e82020-11-25T01:25:40ZengElsevierData in Brief2352-34092020-08-0131105765Data on the evaluation of FGF2 gene expression in Colorectal CancerHelena Caiado0Natércia Conceição1Daniel Tiago2Ana Marreiros3Susana Vicente4Jose Luis Enriquez5Ana Margarida Vaz6Artur Antunes7Horácio Guerreiro8Paulo Caldeira9M. Leonor Cancela10ProRegeM PhD Programme in Mechanisms of disease and regenerative medicine, University of Algarve, Faro, 8005-139, Portugal; Center of Marine Sciences (CCMAR), University of Algarve, Faro, 8005-139, Portugal; Department of Biomedical Sciences and Medicine, University of Algarve, Faro, 8005-139, PortugalCenter of Marine Sciences (CCMAR), University of Algarve, Faro, 8005-139, Portugal; Department of Biomedical Sciences and Medicine, University of Algarve, Faro, 8005-139, Portugal; Algarve Biomedical Center, University of Algarve, Faro, 8005-139, Portugal; Corresponding author.Center of Marine Sciences (CCMAR), University of Algarve, Faro, 8005-139, Portugal; Residency at University Hospital of Algarve, Faro 8000-386Department of Biomedical Sciences and Medicine, University of Algarve, Faro, 8005-139, Portugal; Algarve Biomedical Center, University of Algarve, Faro, 8005-139, PortugalPathology Department, University Hospital of Algarve, Faro, 8000-386Pathology Department, University Hospital of Algarve, Faro, 8000-386Gastroenterology Department, University Hospital of Algarve, Faro, 8000-386Gastroenterology Department, University Hospital of Algarve, Faro, 8000-386Gastroenterology Department, University Hospital of Algarve, Faro, 8000-386Department of Biomedical Sciences and Medicine, University of Algarve, Faro, 8005-139, Portugal; Gastroenterology Department, University Hospital of Algarve, Faro, 8000-386Center of Marine Sciences (CCMAR), University of Algarve, Faro, 8005-139, Portugal; Department of Biomedical Sciences and Medicine, University of Algarve, Faro, 8005-139, Portugal; Algarve Biomedical Center, University of Algarve, Faro, 8005-139, Portugal; Centre for Biomedical Research, University of Algarve, Faro 8000-139, Portugal; Corresponding author.The data presented in this article is related with the research paper entitled “Evaluation of MGP gene expression in colorectal cancer”, available on Gene journal [1]. From all the transcription factors known to regulate MGP, FGF2 is the most described in colon adenocarcinoma and colon tumor cell lines, where it was shown to: i) contribute for the invasiveness potential; and ii) promote proliferation and survival of colorectal cancer cells. These in vitro studies pose the hypothesis that FGF2 associated signaling pathways could be promoting the regulation of others genes, such as MGP, that may lead to tumor progression which ultimately could result in poor prognosis in colon adenocarcinoma.http://www.sciencedirect.com/science/article/pii/S2352340920306594Colorectal Cancer (CRC)Matrix gla proteinGene expressionTranscription factorsFGF2
collection DOAJ
language English
format Article
sources DOAJ
author Helena Caiado
Natércia Conceição
Daniel Tiago
Ana Marreiros
Susana Vicente
Jose Luis Enriquez
Ana Margarida Vaz
Artur Antunes
Horácio Guerreiro
Paulo Caldeira
M. Leonor Cancela
spellingShingle Helena Caiado
Natércia Conceição
Daniel Tiago
Ana Marreiros
Susana Vicente
Jose Luis Enriquez
Ana Margarida Vaz
Artur Antunes
Horácio Guerreiro
Paulo Caldeira
M. Leonor Cancela
Data on the evaluation of FGF2 gene expression in Colorectal Cancer
Data in Brief
Colorectal Cancer (CRC)
Matrix gla protein
Gene expression
Transcription factors
FGF2
author_facet Helena Caiado
Natércia Conceição
Daniel Tiago
Ana Marreiros
Susana Vicente
Jose Luis Enriquez
Ana Margarida Vaz
Artur Antunes
Horácio Guerreiro
Paulo Caldeira
M. Leonor Cancela
author_sort Helena Caiado
title Data on the evaluation of FGF2 gene expression in Colorectal Cancer
title_short Data on the evaluation of FGF2 gene expression in Colorectal Cancer
title_full Data on the evaluation of FGF2 gene expression in Colorectal Cancer
title_fullStr Data on the evaluation of FGF2 gene expression in Colorectal Cancer
title_full_unstemmed Data on the evaluation of FGF2 gene expression in Colorectal Cancer
title_sort data on the evaluation of fgf2 gene expression in colorectal cancer
publisher Elsevier
series Data in Brief
issn 2352-3409
publishDate 2020-08-01
description The data presented in this article is related with the research paper entitled “Evaluation of MGP gene expression in colorectal cancer”, available on Gene journal [1]. From all the transcription factors known to regulate MGP, FGF2 is the most described in colon adenocarcinoma and colon tumor cell lines, where it was shown to: i) contribute for the invasiveness potential; and ii) promote proliferation and survival of colorectal cancer cells. These in vitro studies pose the hypothesis that FGF2 associated signaling pathways could be promoting the regulation of others genes, such as MGP, that may lead to tumor progression which ultimately could result in poor prognosis in colon adenocarcinoma.
topic Colorectal Cancer (CRC)
Matrix gla protein
Gene expression
Transcription factors
FGF2
url http://www.sciencedirect.com/science/article/pii/S2352340920306594
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