Post-myocardial infarction heart failure dysregulates the bone vascular niche
Hematopoiesis influences the progression of cardiovascular disease, yet the influence of cardiovascular disease on the bone vasculature is unknown. Hoffmann, Luxán, Abplanalp et al. describe the response of the bone cell composition to myocardial infarction and provide a rationale for using anti-inf...
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2021-06-01
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Series: | Nature Communications |
Online Access: | https://doi.org/10.1038/s41467-021-24045-4 |
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doaj-398da0beec4046aba06db75788ef5a982021-06-27T11:12:24ZengNature Publishing GroupNature Communications2041-17232021-06-0112111110.1038/s41467-021-24045-4Post-myocardial infarction heart failure dysregulates the bone vascular nicheJedrzej Hoffmann0Guillermo Luxán1Wesley Tyler Abplanalp2Simone-Franziska Glaser3Tina Rasper4Ariane Fischer5Marion Muhly-Reinholz6Michael Potente7Birgit Assmus8David John9Andreas Michael Zeiher10Stefanie Dimmeler11Department of Cardiology, Center of Internal Medicine, Goethe University FrankfurtGerman Center for Cardiovascular Research DZHKGerman Center for Cardiovascular Research DZHKGerman Center for Cardiovascular Research DZHKInstitute of Cardiovascular Regeneration, Center of Molecular Medicine, Goethe University FrankfurtInstitute of Cardiovascular Regeneration, Center of Molecular Medicine, Goethe University FrankfurtInstitute of Cardiovascular Regeneration, Center of Molecular Medicine, Goethe University FrankfurtAngiogenesis and Metabolism Laboratory, Max Planck Institute for Heart and Lung ResearchDepartment of Cardiology, Center of Internal Medicine, Goethe University FrankfurtGerman Center for Cardiovascular Research DZHKDepartment of Cardiology, Center of Internal Medicine, Goethe University FrankfurtGerman Center for Cardiovascular Research DZHKHematopoiesis influences the progression of cardiovascular disease, yet the influence of cardiovascular disease on the bone vasculature is unknown. Hoffmann, Luxán, Abplanalp et al. describe the response of the bone cell composition to myocardial infarction and provide a rationale for using anti-inflammatory therapies to prevent the deterioration of the bone vascular nichehttps://doi.org/10.1038/s41467-021-24045-4 |
collection |
DOAJ |
language |
English |
format |
Article |
sources |
DOAJ |
author |
Jedrzej Hoffmann Guillermo Luxán Wesley Tyler Abplanalp Simone-Franziska Glaser Tina Rasper Ariane Fischer Marion Muhly-Reinholz Michael Potente Birgit Assmus David John Andreas Michael Zeiher Stefanie Dimmeler |
spellingShingle |
Jedrzej Hoffmann Guillermo Luxán Wesley Tyler Abplanalp Simone-Franziska Glaser Tina Rasper Ariane Fischer Marion Muhly-Reinholz Michael Potente Birgit Assmus David John Andreas Michael Zeiher Stefanie Dimmeler Post-myocardial infarction heart failure dysregulates the bone vascular niche Nature Communications |
author_facet |
Jedrzej Hoffmann Guillermo Luxán Wesley Tyler Abplanalp Simone-Franziska Glaser Tina Rasper Ariane Fischer Marion Muhly-Reinholz Michael Potente Birgit Assmus David John Andreas Michael Zeiher Stefanie Dimmeler |
author_sort |
Jedrzej Hoffmann |
title |
Post-myocardial infarction heart failure dysregulates the bone vascular niche |
title_short |
Post-myocardial infarction heart failure dysregulates the bone vascular niche |
title_full |
Post-myocardial infarction heart failure dysregulates the bone vascular niche |
title_fullStr |
Post-myocardial infarction heart failure dysregulates the bone vascular niche |
title_full_unstemmed |
Post-myocardial infarction heart failure dysregulates the bone vascular niche |
title_sort |
post-myocardial infarction heart failure dysregulates the bone vascular niche |
publisher |
Nature Publishing Group |
series |
Nature Communications |
issn |
2041-1723 |
publishDate |
2021-06-01 |
description |
Hematopoiesis influences the progression of cardiovascular disease, yet the influence of cardiovascular disease on the bone vasculature is unknown. Hoffmann, Luxán, Abplanalp et al. describe the response of the bone cell composition to myocardial infarction and provide a rationale for using anti-inflammatory therapies to prevent the deterioration of the bone vascular niche |
url |
https://doi.org/10.1038/s41467-021-24045-4 |
work_keys_str_mv |
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