Long-Lasting Effect of Infant Rats Endotoxemia on Heat Shock Protein 60 in the Pancreatic Acinar Cells: Involvement of Toll-Like Receptor 4

Long-Lasting Effect of Infant Rats Endotoxemia on Heat Shock Protein 60 in the Pancreatic Acinar Cells: Involvement of Toll-Like Receptor 4

Introduction. Lipopolysaccharide endotoxin (LPS) is responsible for septic shock and multiorgan failure, but pretreatment of rats with low doses of LPS reduced pancreatic acute damage. Aim. We investigated the effects of the endotoxemia induced in the early period of life on Toll-like receptor 4 (TL...

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Bibliographic Details
Main Authors:	Joanna Bonior, Jolanta Jaworek, Michalina Kot, Stanisław J. Konturek, Piotr Pierzchalski
Format:	Article
Language:	English
Published:	Hindawi Limited 2012-01-01
Series:	International Journal of Inflammation
Online Access:	http://dx.doi.org/10.1155/2012/354904

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