Increased oral sodium chloride intake in humans amplifies selectively postprandial GLP‐1 but not GIP, CCK, and gastrin in plasma

Increased oral sodium chloride intake in humans amplifies selectively postprandial GLP‐1 but not GIP, CCK, and gastrin in plasma

Abstract Human studies have demonstrated that physiologically relevant changes in circulating glucagon‐like peptide‐1 (GLP‐1) elicit a rapid increase in renal sodium excretion when combined with expansion of the extracellular fluid volume. Other studies support the involvement of various gastrointes...

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Bibliographic Details
Main Authors: Ali Asmar, Per K. Cramon, Meena Asmar, Lene Simonsen, Charlotte M. Sorensen, Sten Madsbad, Cedric Moro, Bolette Hartmann, Jens F. Rehfeld, Jens J. Holst, Peter Hovind, Boye L. Jensen, Jens Bülow
Format: Article
Language:English
Published: Wiley 2020-08-01
Series:Physiological Reports
Subjects:
Glucagon‐like peptide‐1
gut
gut‐kidney axis
kidney
the renin‐angiotensin‐aldosterone system
Online Access:https://doi.org/10.14814/phy2.14519

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