Juvenile Bow Hunter's Stroke without Hemodynamic Changes
Bow hunter's stroke (BHS) is a cerebrovascular disease caused by occlusion of the vertebral artery (VA) on head rotation. BHS is generally associated with hemodynamic changes, often leading to vertebrobasilar insufficiency symptoms, such as vertigo and faintness. Although artery-to-artery embol...
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doaj-65ee5c2cbcda4108a63775b67029c54b2020-11-25T04:01:10ZengSAGE PublishingClinical Medicine Insights: Case Reports1179-54762010-01-01310.4137/CCRep.S3555Juvenile Bow Hunter's Stroke without Hemodynamic ChangesKozue Saito0Makito Hirano1Toshiaki Taoka2Hiroyuki Nakagawa3Takanori Kitauchi4Masanori Ikeda5Emi Tanizawa6Kimihiko Kichikawa7Satoshi Ueno8Departments of Neurology, Nara Medical University, Kashihara, Nara, Japan.Departments of Neurology, Nara Medical University, Kashihara, Nara, Japan.Departments of Radiology, Nara Medical University, Kashihara, Nara, Japan.Departments of Radiology, Nara Medical University, Kashihara, Nara, Japan.Departments of Neurology, Nara Medical University, Kashihara, Nara, Japan.Departments of Neurology, Nara Medical University, Kashihara, Nara, Japan.Departments of Neurology, Nara Medical University, Kashihara, Nara, Japan.Departments of Radiology, Nara Medical University, Kashihara, Nara, Japan.Departments of Neurology, Nara Medical University, Kashihara, Nara, Japan.Bow hunter's stroke (BHS) is a cerebrovascular disease caused by occlusion of the vertebral artery (VA) on head rotation. BHS is generally associated with hemodynamic changes, often leading to vertebrobasilar insufficiency symptoms, such as vertigo and faintness. Although artery-to-artery embolism has also been proposed as an underlying mechanism, it remains controversial. This report documents a case of BHS without hemodynamic changes. We describe a 26-year-old male patient who had VA occlusion on head rotation and repetitive infarction of thalami. He had an anomalous bypass of the VA and therefore no symptomatic hemodynamic changes. Thus, non-hemodynamic BHS should be considered in juvenile patients with vertebrobasilar stroke.https://doi.org/10.4137/CCRep.S3555 |
collection |
DOAJ |
language |
English |
format |
Article |
sources |
DOAJ |
author |
Kozue Saito Makito Hirano Toshiaki Taoka Hiroyuki Nakagawa Takanori Kitauchi Masanori Ikeda Emi Tanizawa Kimihiko Kichikawa Satoshi Ueno |
spellingShingle |
Kozue Saito Makito Hirano Toshiaki Taoka Hiroyuki Nakagawa Takanori Kitauchi Masanori Ikeda Emi Tanizawa Kimihiko Kichikawa Satoshi Ueno Juvenile Bow Hunter's Stroke without Hemodynamic Changes Clinical Medicine Insights: Case Reports |
author_facet |
Kozue Saito Makito Hirano Toshiaki Taoka Hiroyuki Nakagawa Takanori Kitauchi Masanori Ikeda Emi Tanizawa Kimihiko Kichikawa Satoshi Ueno |
author_sort |
Kozue Saito |
title |
Juvenile Bow Hunter's Stroke without Hemodynamic Changes |
title_short |
Juvenile Bow Hunter's Stroke without Hemodynamic Changes |
title_full |
Juvenile Bow Hunter's Stroke without Hemodynamic Changes |
title_fullStr |
Juvenile Bow Hunter's Stroke without Hemodynamic Changes |
title_full_unstemmed |
Juvenile Bow Hunter's Stroke without Hemodynamic Changes |
title_sort |
juvenile bow hunter's stroke without hemodynamic changes |
publisher |
SAGE Publishing |
series |
Clinical Medicine Insights: Case Reports |
issn |
1179-5476 |
publishDate |
2010-01-01 |
description |
Bow hunter's stroke (BHS) is a cerebrovascular disease caused by occlusion of the vertebral artery (VA) on head rotation. BHS is generally associated with hemodynamic changes, often leading to vertebrobasilar insufficiency symptoms, such as vertigo and faintness. Although artery-to-artery embolism has also been proposed as an underlying mechanism, it remains controversial. This report documents a case of BHS without hemodynamic changes. We describe a 26-year-old male patient who had VA occlusion on head rotation and repetitive infarction of thalami. He had an anomalous bypass of the VA and therefore no symptomatic hemodynamic changes. Thus, non-hemodynamic BHS should be considered in juvenile patients with vertebrobasilar stroke. |
url |
https://doi.org/10.4137/CCRep.S3555 |
work_keys_str_mv |
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