A mathematical model of CR3/TLR2 crosstalk in the context of Francisella tularensis infection.
Complement Receptor 3 (CR3) and Toll-like Receptor 2 (TLR2) are pattern recognition receptors expressed on the surface of human macrophages. Although these receptors are essential components for recognition by the innate immune system, pathogen coordinated crosstalk between them can suppress the pro...
Main Authors: | Rachel Leander, Shipan Dai, Larry S Schlesinger, Avner Friedman |
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Format: | Article |
Language: | English |
Published: |
Public Library of Science (PLoS)
2012-01-01
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Series: | PLoS Computational Biology |
Online Access: | https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/23133361/pdf/?tool=EBI |
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