232 The epithelial-to-mesenchymal transition (EMT) contributes to immunosuppression in breast carcinomas and regulates their response to immune checkpoint blockade
Main Authors: | Stefani Spranger, Maria Zagorulya, Anushka Dongre, Robert Weinberg, Mohammad Rashidian, Elinor Eaton, Ferenc Reinhardt, Prat Thiru, Sunita Nepal, Tuba Banaz, Anna Martner |
---|---|
Format: | Article |
Language: | English |
Published: |
BMJ Publishing Group
2020-11-01
|
Series: | Journal for ImmunoTherapy of Cancer |
Similar Items
-
Epithelial-to-Mesenchymal Transition Contributes to Immunosuppression in Breast Carcinomas
by: Rashidian, Mohammad, et al.
Published: (2018) -
Distinct EMT programs control normal mammary stem cells and tumour-initiating cells
by: Ye, Xin, et al.
Published: (2018) -
Inflammation Triggers Zeb1-Dependent Escape from Tumor Latency
by: Shibue, Tsukasa, et al.
Published: (2018) -
Acquisition of a hybrid E/M state is essential for tumorigenicity of basal breast cancer cells
by: Kröger, Cornelia, et al.
Published: (2020) -
First limits on double beta decays in $$^\mathbf{232}$$ 232 Th
by: M. Laubenstein, et al.
Published: (2020-08-01)