Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition

Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition

Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the origin...

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Main Authors: Tomas Rajtik, Slavka Carnicka, Adrian Szobi, Zoltan Giricz, Jin O-Uchi, Veronika Hassova, Pavel Svec, Peter Ferdinandy, Tanya Ravingerova, Adriana Adameova
Format: Article
Language:English
Published: Elsevier 2016-06-01
Series:Data in Brief
Online Access:http://www.sciencedirect.com/science/article/pii/S2352340916301251
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spelling doaj-7a49268a8081425485b908240be473ac2020-11-25T01:40:29ZengElsevierData in Brief2352-34092016-06-017730734Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibitionTomas Rajtik0Slavka Carnicka1Adrian Szobi2Zoltan Giricz3Jin O-Uchi4Veronika Hassova5Pavel Svec6Peter Ferdinandy7Tanya Ravingerova8Adriana Adameova9Department of Pharmacology and Toxicology, Faculty of Pharmacy, Comenius University in Bratislava, Slovak RepublicInstitute for Heart Research, Slovak Academy of Sciences & Centre of Excellence, SAS NOREG, Bratislava, Slovak RepublicDepartment of Pharmacology and Toxicology, Faculty of Pharmacy, Comenius University in Bratislava, Slovak RepublicCardiometabolic Research Group, Department of Pharmacology and Pharmacotherapy, Semmelweis University, Budapest, HungaryDepartment of Medicine, Jefferson Medical College, Thomas Jefferson University, Philadelphia, USADepartment of Pharmacology and Toxicology, Faculty of Pharmacy, Comenius University in Bratislava, Slovak RepublicDepartment of Pharmacology and Toxicology, Faculty of Pharmacy, Comenius University in Bratislava, Slovak RepublicCardiometabolic Research Group, Department of Pharmacology and Pharmacotherapy, Semmelweis University, Budapest, Hungary; PharmaHungary Group, Szeged, HungaryInstitute for Heart Research, Slovak Academy of Sciences & Centre of Excellence, SAS NOREG, Bratislava, Slovak RepublicDepartment of Pharmacology and Toxicology, Faculty of Pharmacy, Comenius University in Bratislava, Slovak Republic; Correspondence to: Department of Pharmacology and Toxicology, Faculty of Pharmacy, Comenius University in Bratislava, Odbojarov 10, Bratislava 832 32, Slovak Republic. Tel.: +421 2 50117 366; fax: +421 2 50 117 100.Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article “Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis” [1]. Keywords: Myocardial ischemia/reperfusion injury, CaMKII inhibition, AT1 receptor blockade, Apoptosis, Necrosishttp://www.sciencedirect.com/science/article/pii/S2352340916301251
collection DOAJ
language English
format Article
sources DOAJ
author Tomas Rajtik
Slavka Carnicka
Adrian Szobi
Zoltan Giricz
Jin O-Uchi
Veronika Hassova
Pavel Svec
Peter Ferdinandy
Tanya Ravingerova
Adriana Adameova
spellingShingle Tomas Rajtik
Slavka Carnicka
Adrian Szobi
Zoltan Giricz
Jin O-Uchi
Veronika Hassova
Pavel Svec
Peter Ferdinandy
Tanya Ravingerova
Adriana Adameova
Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition
Data in Brief
author_facet Tomas Rajtik
Slavka Carnicka
Adrian Szobi
Zoltan Giricz
Jin O-Uchi
Veronika Hassova
Pavel Svec
Peter Ferdinandy
Tanya Ravingerova
Adriana Adameova
author_sort Tomas Rajtik
title Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition
title_short Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition
title_full Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition
title_fullStr Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition
title_full_unstemmed Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition
title_sort data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of camkii and angiotensin at1 receptor inhibition
publisher Elsevier
series Data in Brief
issn 2352-3409
publishDate 2016-06-01
description Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article “Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis” [1]. Keywords: Myocardial ischemia/reperfusion injury, CaMKII inhibition, AT1 receptor blockade, Apoptosis, Necrosis
url http://www.sciencedirect.com/science/article/pii/S2352340916301251
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