Liraglutide Ameliorates β-Amyloid Deposits and Secondary Damage in the Ipsilateral Thalamus and Sensory Deficits After Focal Cerebral Infarction in Rats

Liraglutide Ameliorates β-Amyloid Deposits and Secondary Damage in the Ipsilateral Thalamus and Sensory Deficits After Focal Cerebral Infarction in Rats

Focal cerebral infarction causes β-amyloid (Aβ) deposition and secondary neuronal degeneration in the ipsilateral thalamus. Thalamus is the subcortical center of sensory, the damage of thalamus could cause sensory deficits. The present study aimed to investigate the protective effects of liraglutide...

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Bibliographic Details
Main Authors: Hui-Li Zhu, Zhang-Pei Liu, Wan-Yong Yang, Da-Wei Dong, Ying Zhao, Bing Yang, Li-An Huang, Yu-Sheng Zhang, An-Ding Xu
Format: Article
Language:English
Published: Frontiers Media S.A. 2018-12-01
Series:Frontiers in Neuroscience
Subjects:
cerebral infarction
glucagon-like peptide-1
liraglutide
β-amyloid
secondary thalamus damage
Online Access:https://www.frontiersin.org/article/10.3389/fnins.2018.00962/full

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