Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART

Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART

Bibliographic Details
Main Authors: J. Symons, C. Bacchus-Souffan, A. Chopra, S. Leary, D. Cameron, P.U. Cameron, R. Hoh, H. Ahn, S.G. Deeks, J.M. McCune, S. Mallal, P.W. Hunt, S.R. Lewin
Format: Article
Language:English
Published: Elsevier 2019-07-01
Series:Journal of Virus Eradication
Online Access:http://www.sciencedirect.com/science/article/pii/S2055664020310736
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spelling doaj-a910fc048d5442caab0629050c6e75462021-05-05T04:07:25ZengElsevierJournal of Virus Eradication2055-66402019-07-0151Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ARTJ. Symons0C. Bacchus-Souffan1A. Chopra2S. Leary3D. Cameron4P.U. Cameron5R. Hoh6H. Ahn7S.G. Deeks8J.M. McCune9S. Mallal10P.W. Hunt11S.R. Lewin12Peter Doherty Institute for Infection and Immunity, University of Melbourne and Royal Melbourne Hospital, Melbourne, AustraliaUniversity of California - San Francisco, Division of Experimental Medicine, Department of Medicine, San Francisco, USAMurdoch University, Institute for Immunology and Infectious Diseases, Murdoch, AustraliaMurdoch University, Institute for Immunology and Infectious Diseases, Murdoch, AustraliaPeter Doherty Institute for Infection and Immunity, University of Melbourne and Royal Melbourne Hospital, Melbourne, AustraliaPeter Doherty Institute for Infection and Immunity, University of Melbourne and Royal Melbourne Hospital, Melbourne, Australia; Alfred Hospital and Monash University, Department of Infectious Diseases, Melbourne, AustraliaUniversity of California - San Francisco, Division of HIV, Infectious Diseases and Global Medicine, Department of Medicine, Zuckerberg San Francisco General Hospital, San Francisco, USAUniversity of California - San Francisco, Division of Experimental Medicine, Department of Medicine, San Francisco, USAUniversity of California - San Francisco, Division of HIV, Infectious diseases and Global Medicine, Department of Medicine, San Francisco, USAUniversity of California - San Francisco, Division of Experimental Medicine, Department of Medicine, San Francisco, USAMurdoch University, Institute for Immunology and Infectious Diseases, Murdoch, Australia; Vanderbilt University Medical Center (VUMC), Department of Medicine, Nashville, USAUniversity of California - San Francisco, Division of Experimental Medicine, Department of Medicine, San Francisco, USAPeter Doherty Institute for Infection and Immunity, University of Melbourne and Royal Melbourne Hospital, Melbourne, Australia; Alfred Hospital and Monash University, Department of Infectious Diseases, Melbourne, Australiahttp://www.sciencedirect.com/science/article/pii/S2055664020310736
collection DOAJ
language English
format Article
sources DOAJ
author J. Symons
C. Bacchus-Souffan
A. Chopra
S. Leary
D. Cameron
P.U. Cameron
R. Hoh
H. Ahn
S.G. Deeks
J.M. McCune
S. Mallal
P.W. Hunt
S.R. Lewin
spellingShingle J. Symons
C. Bacchus-Souffan
A. Chopra
S. Leary
D. Cameron
P.U. Cameron
R. Hoh
H. Ahn
S.G. Deeks
J.M. McCune
S. Mallal
P.W. Hunt
S.R. Lewin
Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART
Journal of Virus Eradication
author_facet J. Symons
C. Bacchus-Souffan
A. Chopra
S. Leary
D. Cameron
P.U. Cameron
R. Hoh
H. Ahn
S.G. Deeks
J.M. McCune
S. Mallal
P.W. Hunt
S.R. Lewin
author_sort J. Symons
title Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART
title_short Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART
title_full Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART
title_fullStr Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART
title_full_unstemmed Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART
title_sort clonal integration site expansion of infected cells is a main contributor of hiv persistence in more differentiated t cell subsets during suppressive art
publisher Elsevier
series Journal of Virus Eradication
issn 2055-6640
publishDate 2019-07-01
url http://www.sciencedirect.com/science/article/pii/S2055664020310736
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