Potential Roles of NIX/BNIP3L Pathway in Rat Traumatic Brain Injury
NIX/BNIP3L is known as a proapoptotic protein that is also related to mitophagy. Previous reports have shown that NIX could be involved in neuronal apoptosis after intracerebral hemorrhage, but it also plays a protective role in mitophagy in ischemic brain injury. How NIX works in traumatic brain in...
Main Authors: | Jialing Ma, Haibo Ni, Qin Rui, Huixiang Liu, Feng Jiang, Rong Gao, Yanping Gao, Di Li, Gang Chen |
---|---|
Format: | Article |
Language: | English |
Published: |
SAGE Publishing
2019-05-01
|
Series: | Cell Transplantation |
Online Access: | https://doi.org/10.1177/0963689719840353 |
Similar Items
-
Annexin A7 Levels Increase in Rats With Traumatic Brain Injury and Promote Secondary Brain Injury
by: Fan Gao, et al.
Published: (2018-05-01) -
LRRK2 Contributes to Secondary Brain Injury Through a p38/Drosha Signaling Pathway After Traumatic Brain Injury in Rats
by: Qin Rui, et al.
Published: (2018-03-01) -
2-BFI Provides Neuroprotection Against Inflammation and Necroptosis in a Rat Model of Traumatic Brain Injury
by: Haibo Ni, et al.
Published: (2019-06-01) -
BNIP3 and NIX mediate Mieap-induced accumulation of lysosomal proteins within mitochondria.
by: Yasuyuki Nakamura, et al.
Published: (2012-01-01) -
The role of KLHL23 in BNIP3L/NIX -mediated mitophagy in Hepatocellular Carcinoma
by: Yu Chen Hung, et al.
Published: (2016)