DNA End Resection: Facts and
DNA double-strand breaks (DSBs), which arise following exposure to a number of endogenous and exogenous agents, can be repaired by either the homologous recombination (HR) or non-homologous end-joining (NHEJ) pathways in eukaryotic cells. A vital step in HR repair is DNA end resection, which generat...
Main Authors: | Ting Liu, Jun Huang |
---|---|
Format: | Article |
Language: | English |
Published: |
Elsevier
2016-06-01
|
Series: | Genomics, Proteomics & Bioinformatics |
Subjects: | |
Online Access: | http://www.sciencedirect.com/science/article/pii/S1672022916300717 |
Similar Items
-
Controlling DNA-end resection: an emerging task for Ubiquitin and SUMO
by: Sarah-Felicitas Himmels, et al.
Published: (2016-08-01) -
Controlling DNA-end resection: a new task for CDKs
by: Lorenza P Ferretti, et al.
Published: (2013-06-01) -
Targeting of the Fun30 nucleosome remodeller by the Dpb11 scaffold facilitates cell cycle-regulated DNA end resection
by: Susanne CS Bantele, et al.
Published: (2017-01-01) -
CtIP forms a tetrameric dumbbell-shaped particle which bridges complex DNA end structures for double-strand break repair
by: Oliver J Wilkinson, et al.
Published: (2019-01-01) -
SCR7, a potent cancer therapeutic agent and a biochemical inhibitor of nonhomologous DNA end‐joining
by: Meghana Manjunath, et al.
Published: (2021-06-01)