"BreakThrough cancer Pain" biomolecular mechanisms
The BTcP is a transitory exacerbation of pain of moderate to high intensity, which occurs, either spontaneously or as a result of a trigger factor, in patients with pain basic maintained for most of the day or under control of mild . The pathophysiological mechanisms underlying the development and...
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2014-11-01
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| Series: | Pathos |
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| Online Access: | http://www.pathos-journal.com/page_79.html |
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doaj-debce4ceba6746d583f278fd2f5ed9022020-11-24T23:50:20ZengPublieditingPathos1593-23541593-23542014-11-01213"BreakThrough cancer Pain" biomolecular mechanisms Francesco Amato0Direttore UOC Anestesia, Terapia del Dolore, CP e Dipartimento di Emergenza AO CosenzaThe BTcP is a transitory exacerbation of pain of moderate to high intensity, which occurs, either spontaneously or as a result of a trigger factor, in patients with pain basic maintained for most of the day or under control of mild . The pathophysiological mechanisms underlying the development and maintenance of this type of pain seem to depend on several factors including an increase in the activity of the receptors TRPV1, central sensitization, activation of Glia etc. To better manage the disease can interfere with rapid analgesia of short duration that best overlaps the temporal characteristics of BTcP. http://www.pathos-journal.com/page_79.htmlBTcPcentral sensitizationGliaReceptors TRPV1 |
| collection |
DOAJ |
| language |
English |
| format |
Article |
| sources |
DOAJ |
| author |
Francesco Amato |
| spellingShingle |
Francesco Amato "BreakThrough cancer Pain" biomolecular mechanisms Pathos BTcP central sensitization Glia Receptors TRPV1 |
| author_facet |
Francesco Amato |
| author_sort |
Francesco Amato |
| title |
"BreakThrough cancer Pain" biomolecular mechanisms |
| title_short |
"BreakThrough cancer Pain" biomolecular mechanisms |
| title_full |
"BreakThrough cancer Pain" biomolecular mechanisms |
| title_fullStr |
"BreakThrough cancer Pain" biomolecular mechanisms |
| title_full_unstemmed |
"BreakThrough cancer Pain" biomolecular mechanisms |
| title_sort |
"breakthrough cancer pain" biomolecular mechanisms |
| publisher |
Publiediting |
| series |
Pathos |
| issn |
1593-2354 1593-2354 |
| publishDate |
2014-11-01 |
| description |
The BTcP is a transitory exacerbation of pain of moderate to high intensity, which occurs, either spontaneously or as a result of a trigger factor, in patients with pain basic maintained for most of the day or under control of mild .
The pathophysiological mechanisms underlying the development and maintenance of this type of pain seem to depend on several factors including an increase in the activity of the receptors TRPV1, central sensitization, activation of Glia etc.
To better manage the disease can interfere with rapid analgesia of short duration that best overlaps the temporal characteristics of BTcP.
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| topic |
BTcP central sensitization Glia Receptors TRPV1 |
| url |
http://www.pathos-journal.com/page_79.html |
| work_keys_str_mv |
AT francescoamato breakthroughcancerpainbiomolecularmechanisms |
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1725479139942596608 |