Subacute Zinc Administration and L-NAME Caused an Increase of NO, Zinc, Lipoperoxidation, and Caspase-3 during a Cerebral Hypoxia-Ischemia Process in the Rat

Subacute Zinc Administration and L-NAME Caused an Increase of NO, Zinc, Lipoperoxidation, and Caspase-3 during a Cerebral Hypoxia-Ischemia Process in the Rat

Zinc or L-NAME administration has been shown to be protector agents, decreasing oxidative stress and cell death. However, the treatment with zinc and L-NAME by intraperitoneal injection has not been studied. The aim of our work was to study the effect of zinc and L-NAME administration on nitrosative...

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Bibliographic Details
Main Authors: Victor Manuel Blanco-Alvarez, Patricia Lopez-Moreno, Guadalupe Soto-Rodriguez, Daniel Martinez-Fong, Hector Rubio, Juan Antonio Gonzalez-Barrios, Celia Piña-Leyva, Maricela Torres-Soto, María de Jesus Gomez-Villalobos, Daniel Hernandez-Baltazar, Eduardo Brambila, José Ramon Eguibar, Araceli Ugarte, Jorge Cebada, Bertha Alicia Leon-Chavez
Format: Article
Language:English
Published: Hindawi Limited 2013-01-01
Series:Oxidative Medicine and Cellular Longevity
Online Access:http://dx.doi.org/10.1155/2013/240560

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