Fixin' to Divide

In this issue of Molecular Cell, Yata et al. (2012) show that the mitotic kinase and cell-cycle regulator Plk1 can directly stimulate the DNA repair process, providing a potential mechanism of crosstalk between DNA repair and cell-cycle signaling.

Bibliographic Details
Main Authors: Mohammad, Duaa H. (Contributor), Yaffe, Michael B (Author), Yaffe, Michael B (Author)
Other Authors: Massachusetts Institute of Technology. Department of Biological Engineering (Contributor), Massachusetts Institute of Technology. Department of Biology (Contributor), Koch Institute for Integrative Cancer Research at MIT (Contributor), Yaffe, Michael B. (Contributor)
Format: Article
Language:English
Published: Elsevier, 2014-11-20T13:39:41Z.
Subjects:
Online Access:Get fulltext
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100 1 0 |a Mohammad, Duaa H.  |e author 
100 1 0 |a Massachusetts Institute of Technology. Department of Biological Engineering  |e contributor 
100 1 0 |a Massachusetts Institute of Technology. Department of Biology  |e contributor 
100 1 0 |a Koch Institute for Integrative Cancer Research at MIT  |e contributor 
100 1 0 |a Yaffe, Michael B.  |e contributor 
100 1 0 |a Mohammad, Duaa H.  |e contributor 
700 1 0 |a Yaffe, Michael B.  |e author 
700 1 0 |a Yaffe, Michael B  |e author 
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520 |a In this issue of Molecular Cell, Yata et al. (2012) show that the mitotic kinase and cell-cycle regulator Plk1 can directly stimulate the DNA repair process, providing a potential mechanism of crosstalk between DNA repair and cell-cycle signaling. 
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773 |t Molecular Cell