Gene Therapy for Endothelial Progenitor Cell Dysfunction
Endothelial progenitor cells (EPCs) have reduced neovascularization capacity in the context of coronary artery disease (CAD) or cardiac risk factors (RFs). Since, endothelial NO synthase (eNOS) is critical to normal EPC function, we hypothesized that bone marrow cells (BMCs) from rats with RFs and E...
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Language: | en_ca |
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2009
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Online Access: | http://hdl.handle.net/1807/19108 |
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