The Role of Prothymosin α in the Regulation of miR-223 Expression in Emphysema
碩士 === 國立成功大學 === 生物化學暨分子生物學研究所 === 102 === Emphysema, a major subtype of chronic obstructive pulmonary disease (COPD), results from histone deacetylase 2 (HDAC2) dysfunction and inflammatory responses. However, the underlying mechanism is still unclear. We have previously demonstrated that the expr...
Main Authors: | Yi-ChiChen, 陳逸琪 |
---|---|
Other Authors: | Chao-Liang Wu |
Format: | Others |
Language: | zh-TW |
Published: |
2014
|
Online Access: | http://ndltd.ncl.edu.tw/handle/tyb4ef |
Similar Items
-
Overexpression of prothymosin α induced by cigarette smoke extract generates pulmonary emphysema
by: Ya-ChiehShiang, et al.
Published: (2012) -
Transgenic overexpression of Prothymosin α induces development of polycystic kidney disease and emphysema
by: Kuo-Jung Li, et al.
Published: (2005) -
Overexpression of prothymosin α induces pulmonary emphysema by inhibition of TGF-β signalling pathway
by: Chih-HauSu, et al.
Published: (2010) -
Potential role of miR-9 and miR-223 in recurrent ovarian cancer
by: McGuinness Eamonn, et al.
Published: (2008-04-01) -
Prothymosinαgenetherapyinmurinebladdercancer
by: Lin, Pei-Ru, et al.