Effects of Two Maximal Isometric Contractions Attenuate Eccentric Exercise-induced Muscle Damage of Knee Flexors and Surface Electromyographic Activity

碩士 === 國立嘉義大學 === 體育與健康休閒學系研究所 === 105 === Background: Tseng et al. (2016) reported that changes in MaxECC-induced muscle damage (EIMD) of the preconditioning MVC training of the knee extensors (KE) for untrained men were significantly smaller than the control group. No studies have recruited untrai...

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Bibliographic Details
Main Authors: Cai, Jia-Jhen, 蔡佳蓁
Other Authors: Lin, Ming-Ju
Format: Others
Online Access:http://ndltd.ncl.edu.tw/handle/q5t3cn
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Summary:碩士 === 國立嘉義大學 === 體育與健康休閒學系研究所 === 105 === Background: Tseng et al. (2016) reported that changes in MaxECC-induced muscle damage (EIMD) of the preconditioning MVC training of the knee extensors (KE) for untrained men were significantly smaller than the control group. No studies have recruited untrained participants, and targeted on hamstring muscle strains in which are the most frequent sporting injuries to clarify whether the non-damaging exercise of MVC can be attenuated muscle damage induced by MaxECC of knee flexors (KF). Purpose: This study investigated the neural factors (surface electromyography, EMG) of two maximal isometric contractions (2MVC) at 20° knee flexion on changes in indirect muscle damage markers following 60 MaxECC of KF performed 2 days later. Methods: Sixteen untrained young males were randomly placed into the control group (CON) that did not perform 2MVC, or the experimental group (2d, n = 8 per group) who performed 2MVC 2 days before MaxECC. Changes in muscle soreness (SOR), non-dominant upper thigh circumference (CIR), resting knee angle (RANG), maximal voluntary isokinetic concentric contraction (MVC-CON), and surface EMG [median frequency (MF), root mean square (RMS)] before, immediately after, and 1-5 days after MaxECC were compared between groups by a mixed-design of two-way ANOVA. Results: 1). Significant (p < .05) changes in some dependent variables after 2MVC compared to baseline for the 2d group; 2). Changes in the muscle damage variables (e.g. MVC-CON: -13.1%; CIR: +1.5 mm) following MaxECC immediately for the 2d group were smaller than CON group (-22.4%; +4.8 mm); 3) Changes in the surface EMG activity following MaxECC for the 2d group showed smaller changes (MF: 60.2 ± 9.1 Hz) than CON group (70 ± 13.2 Hz). Conclusion: These results suggest that protective effect conferred by the non-damaging exercise of 2MVC against subsequent MaxECC-induced muscle damage is likely to be related to neural adaptations. Therefore, the RBE protocol of this study may provide some useful information for men to minimize muscle damage when they start to participate exercise. It is also required to further understand the underpinning mechanisms of the repeated bout effect in both physiological and pathological contexts.