Enhanced strength development on seaweed supplementovariectomized rats

碩士 === 臺北市立大學 === 運動科學研究所 === 107 === Background: Post-menopause population suffer from sarcopenia which causes decreased muscle mass and strength. Furthermore, loss of muscle mass and decrease of strength also hamper the quality of life of female elderly. Resistance training is an effective way to...

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Bibliographic Details
Main Authors: Lu, Chi-Hong, 呂季鴻
Other Authors: Kuo, Chia-Hua
Format: Others
Language:en_US
Published: 2019
Online Access:http://ndltd.ncl.edu.tw/handle/xr7c78
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Summary:碩士 === 臺北市立大學 === 運動科學研究所 === 107 === Background: Post-menopause population suffer from sarcopenia which causes decreased muscle mass and strength. Furthermore, loss of muscle mass and decrease of strength also hamper the quality of life of female elderly. Resistance training is an effective way to ameliorate the condition above. In addition, seaweed is a world-wide supplement, which is briefly studied that this marine herbal could help insulin sensitivity which allow cells to activate synthesis of protein. Since, we want to test whether combine these interventions could further enhance strength performance. Method: Fourteen ovariectomized rats were separated into either exercise or exercise-seaweed group. All rats received a 10-week resistance training which operated in weight-hanging ladder climbing model. ES group were gavaged with seaweed after every training session. Body composition and oral glucose tolerance was tested. Strength performance was recorded each training session. Blood samples were collected to determine insulin level. Result: Both E and ES receive muscle mass improvement (33 ± 3.7 g; 39 ± 13 g, E and ES, respectively) and body fat percentage loss ( 9 ± 0.5 %; 8 ± 2.9 %, E and ES, respectively). ES develop strength to 1.7 times and 1.9 times of initial record faster than E. Fasting blood glucose and 60-min after OGTT is higher in ES compared to E. Fasting insulin is higher in ES compared to E. We concluded that seaweed could help the force development, yet the insulin might not be the underlying mechanism.