Kaang Bong-kiun

| mr = Kang Ponggyun | rr = Gang Bong-gyun }}

Kaang Bong-Kiun (born November 21, 1961) is a South Korean professor of neuroscience in the Department of Biological Sciences of Seoul National University. He is a fellow of the Korean Academy of Science and Technology and co-director of the IBS Center for Cognition and Sociality with Changjoon Justin Lee. Provided by Wikipedia
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  1. 1
    Protein degradation during reconsolidation as a mechanism for memory reorganization
    by Bong-Kiun Kaang, Bong-Kiun Kaang, Jun-Hyeok Choi
    Published 2011-02-01
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  2. 2
    A novel analytic measure for the water maze utilizing the concept of entropy
    by Hye Ryeon Lee, Bong-Kiun Kaang
    Published 2010-05-01
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  3. 3
    Elevated contextual fear memory by SIRT6 depletion in excitatory neurons of mouse forebrain
    by Hyopil Kim, Hyun-Seok Kim, Bong-Kiun Kaang
    Published 2018-09-01
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  4. 4
    Overexpression of activated CaMKII in the CA1 hippocampus impairs context discrimination, but not contextual conditioning
    by Sanghyun Ye, Ji-il Kim, Jooyoung Kim, Bong-Kiun Kaang
    Published 2019-04-01
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  5. 5
    Further evidence that CP-AMPARs are critically involved in synaptic tag and capture at hippocampal CA1 synapses
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  6. 6
    Proteomic analysis of synaptic protein turnover in the anterior cingulate cortex after nerve injury
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  7. 7
    Superresolution fluorescence microscopy for 3D reconstruction of thick samples
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  8. 8
    Differential sensitivity of three forms of hippocampal synaptic potentiation to depotentiation
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  9. 9
    Conditional knock out of transcription factor CTCF in excitatory neurons induces cognitive deficiency
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  10. 10
    Increased PKMζ activity impedes lateral movement of GluA2-containing AMPA receptors
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  11. 11
    Identification of postsynaptic phosphatidylinositol-4,5-bisphosphate (PIP2) roles for synaptic plasticity using chemically induced dimerization
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  12. 12
    In memoriam: John Lisman – commentaries on CaMKII as a memory molecule
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  13. 13
    Elevated RalA activity in the hippocampus of PI3Kγ knock-out mice lacking NMDAR-dependent long-term depression
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  14. 14
    On the Role of Calcium-Permeable AMPARs in Long-Term Potentiation and Synaptic Tagging in the Rodent Hippocampus
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  15. 15
    PKA drives an increase in AMPA receptor unitary conductance during LTP in the hippocampus
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  16. 16
    Identification of a novel Shank2 transcriptional variant in Shank2 knockout mouse model of autism spectrum disorder
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  17. 17
    Autophagy pathway upregulation in a human iPSC-derived neuronal model of Cohen syndrome with VPS13B missense mutations
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  18. 18
    Cohen Syndrome Patient iPSC-Derived Neurospheres and Forebrain-Like Glutamatergic Neurons Reveal Reduced Proliferation of Neural Progenitor Cells and Altered Expression of Synapse Genes
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  19. 19
    The Probability of Neurotransmitter Release Governs AMPA Receptor Trafficking via Activity-Dependent Regulation of mGluR1 Surface Expression
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  20. 20
    The Role of Calcium-Permeable AMPARs in Long-Term Potentiation at Principal Neurons in the Rodent Hippocampus
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