H. Robert Horvitz

Horvitz in 2003 Howard Robert Horvitz ForMemRS NAS AAA&S APS NAM (born May 8, 1947) is an American biologist best known for his research on the nematode worm ''Caenorhabditis elegans'', for which he was awarded the 2002 Nobel Prize in Physiology or Medicine, together with Sydney Brenner and John E. Sulston, whose "seminal discoveries concerning the genetic regulation of organ development and programmed cell death" were "important for medical research and have shed new light on the pathogenesis of many diseases". Provided by Wikipedia
Showing 1 - 20 results of 49 for search 'Horvitz, Howard Robert', query time: 0.19s Refine Results
  1. 1
    Variants of the elongator protein 3 (ELP3) gene are associated with motor neuron degeneration
    by Horvitz, Howard Robert
    Published 2011
    Get fulltext
    Article
  2. 2
    The Translational Regulators GCN-1 and ABCF-3 Act Together to Promote Apoptosis in C. elegans
    Get fulltext
    Article
  3. 3
    The C. elegans MicroRNA mir-71 Acts in Neurons to Promote Germline-Mediated Longevity through Regulation of DAF-16/FOXO
    Get fulltext
    Article
  4. 4
    Light and Hydrogen Peroxide Inhibit C. elegans Feeding through Gustatory Receptor Orthologs and Pharyngeal Neurons
    Other Authors: “...Horvitz, Howard Robert...”
    Get fulltext
    Article
  5. 5
    The CDK8 Complex and Proneural Proteins Together Drive Neurogenesis from a Mesodermal Lineage
    by Luo, Shuo, Horvitz, Howard Robert
    Published 2018
    Other Authors: “...Horvitz, Howard Robert...”
    Get fulltext
    Article
  6. 6
    Mutations in the Caenorhabditis elegans U2AF Large Subunit UAF-1 Al= of a 3' Splice Site In Vivo
    by Ma, Long, Horvitz, Howard Robert
    Published 2010
    Get fulltext
    Article
  7. 7
    Mutations in the Caenorhabditis elegans U2AF Large Subunit UAF-1 Alter the Choice of a 3' Splice Site In Vivo
    by Ma, Long, Horvitz, Howard Robert
    Published 2010
    Get fulltext
    Article
  8. 8
    An Sp1 transcription factor coordinates caspase-dependent and -independent apoptotic pathways
    Get fulltext
    Article
  9. 9
    Many families of Caenorhabditis elegans microRNAs are not essential for development or viability
    Get fulltext
    Article
  10. 10
    Reduced expression of the Kinesin-Associated Protein 3 (KIFAP3) gene increases survival in sporadic amyotrophic lateral sclerosis
    Get fulltext
    Article
  11. 11
    Both the apoptotic suicide pathway and phagocytosis are required for a programmed cell death in Caenorhabditis elegans
    Get fulltext
    Article
  12. 12
    Hypoxia-inducible factor cell non-autonomously regulates C. elegans stress responses and behavior via a nuclear receptor
    Get fulltext
    Article
  13. 13
    A C9orf72 ALS/FTD Ortholog Acts in Endolysosomal Degradation and Lysosomal Homeostasis
    Get fulltext
    Article
  14. 14
    Replication-Coupled Chromatin Assembly Generates a Neuronal Bilateral Asymmetry in C. elegans
    Get fulltext
    Article
  15. 15
    Ligand-Gated Chloride Channels Are Receptors for Biogenic Amines in C. elegans
    Get fulltext
    Article
  16. 16
    Six and Eya promote apoptosis through direct transcriptional activation of the proapoptotic BH3-only gene egl-1 in Caenorhabditis elegans
    Get fulltext
    Article
  17. 17
    Programmed elimination of cells by caspase-independent cell extrusion in C. elegans
    Get fulltext
    Article
  18. 18
    Both the Caspase CSP-1 and a Caspase-Independent Pathway Promote Programmed Cell Death in Parallel to the Canonical Pathway for Apoptosis in Caenorhabditis elegans
    Get fulltext
    Article
  19. 19
    CYSL-1 Interacts with the O[subscript 2]-Sensing Hydroxylase EGL-9 to Promote H[subscript 2]S-Modulated Hypoxia-Induced Behavioral Plasticity in C. elegans
    Get fulltext
    Article
  20. 20
    SLI-1 Cbl Inhibits the Engulfment of Apoptotic Cells in C. elegans through a Ligase-Independent Function
    Get fulltext
    Article
Search Tools: Get RSS Feed — Email this Search