Pablo Caviedes

Caviedes in 2021 Pablo Caviedes was born in 1971 in Cotacachi, Ecuador and lives and works in New York City. He is a multidisciplinary artist who works across various media such as painting, video art, drawing, etching, installation and sculpture. He is considered part of the 21st Century US-Latino transcultural art-scene. His work focuses on social issues such as immigration, displacement and identity.

He has exhibited in Ecuador, France, Spain, Poland, Italy, Puerto Rico, Argentina and the United States.

He studied at the Instituto Superior Tecnológico de Artes Plásticas Daniel Reyes in San Antonio de Ibarra, Ecuador and at the École Nationale Supérieure des Beaux-Arts, in Paris, France''.'' Provided by Wikipedia
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    Altered calcium currents in cultured sensory neurons of normal and trisomy 16 mouse fetuses, an animal model for human trisomy 21 (Down Syndrome)
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    Criopreservación de células estromales derivadas del tejido adiposo: viabilidad utilizando un medio definido libre de proteína animal
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    RCAN1 Knockdown Reverts Defects in the Number of Calcium-Induced Exocytotic Events in a Cellular Model of Down Syndrome
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    Correction to: Microencapsulation of cellular aggregates composed of differentiated insulin and glucagon-producing cells from human mesenchymal stem cells derived from adipose tissue
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    Microencapsulation of cellular aggregates composed of differentiated insulin and glucagon-producing cells from human mesenchymal stem cells derived from adipose tissue
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    Comparative study of the neural differentiation capacity of mesenchymal stromal cells from different tissue sources: An approach for their use in neural regeneration therapies.
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    Small-molecule aggregation inhibitors reduce excess amyloid in a trisomy 16 mouse cortical cell line
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    The Effect of the Nanoscale Structure of Nanobioceramics on Their In Vitro Bioactivity and Cell Differentiation Properties
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    <i>N</i>-Acetylcysteine Reduces Skeletal Muscles Oxidative Stress and Improves Grip Strength in Dysferlin-Deficient Bla/J Mice
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    On the neurotoxicity mechanism of leukoaminochrome o-semiquinone radical derived from dopamine oxidation: mitochondria damage, necrosis, and hydroxyl radical formation
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    Dynamin-2 mutations linked to Centronuclear Myopathy impair actin-dependent trafficking in muscle cells
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