Pulmonary hypertension in wild type mice and animals with genetic deficit in KCa2.3 and KCa3.1 channels.

Pulmonary hypertension in wild type mice and animals with genetic deficit in KCa2.3 and KCa3.1 channels.

In vascular biology, endothelial KCa2.3 and KCa3.1 channels contribute to arterial blood pressure regulation by producing membrane hyperpolarization and smooth muscle relaxation. The role of KCa2.3 and KCa3.1 channels in the pulmonary circulation is not fully established. Using mice with genetically...

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Bibliographic Details
Main Authors: Christine Wandall-Frostholm, Lykke Moran Skaarup, Veeranjaneyulu Sadda, Gorm Nielsen, Elise Røge Hedegaard, Susie Mogensen, Ralf Köhler, Ulf Simonsen
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2014-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC4032241?pdf=render

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http://europepmc.org/articles/PMC4032241?pdf=render

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