Genetic KCa3.1-deficiency produces locomotor hyperactivity and alterations in cerebral monoamine levels.

The calmodulin/calcium-activated K(+) channel KCa3.1 is expressed in red and white blood cells, epithelia and endothelia, and possibly central and peripheral neurons. However, our knowledge about its contribution to neurological functions and behavior is incomplete. Here, we investigated whether gen...

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Bibliographic Details
Main Authors: Kate Lykke Lambertsen, Jan Bert Gramsbergen, Mithula Sivasaravanaparan, Nicholas Ditzel, Linda Maria Sevelsted-Møller, Aida Oliván-Viguera, Maj Rabjerg, Heike Wulff, Ralf Köhler
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2012-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC3471871?pdf=render