Addition of a sequence from α<sub>2</sub>-antiplasmin transforms human serum albumin into a blood clot component that speeds clot lysis
<p>Abstract</p> <p>Background</p> <p>The plasma protein α<sub>2</sub>-antiplasmin (α<sub>2</sub>AP) is cross-linked to fibrin in blood clots by the transglutaminase factor XIIIa, and in that location retards clot lysis. Competition for this effec...
| Main Authors: | , , , |
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| Format: | Article |
| Language: | English |
| Published: |
BMC
2009-03-01
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| Series: | BMC Biotechnology |
| Online Access: | http://www.biomedcentral.com/1472-6750/9/15 |