Development of experimental chronic kidney disease and vascular calcification alters diurnal variation of phosphate and its hormonal regulators

Development of experimental chronic kidney disease and vascular calcification alters diurnal variation of phosphate and its hormonal regulators

Abstract The mineral‐bone axis is tightly regulated and dependent on renal function. In chronic kidney disease (CKD) progressive loss of renal capacity disrupts this axis over‐time, with marked changes in circulating calcium, phosphate, PTH, and fibroblast growth factor‐23 (FGF‐23). These changes co...

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Bibliographic Details
Main Authors: Bruno A. Svajger, Justin L. H. Riddoch, Cynthia M. Pruss, Kimberly J. Laverty, Emilie Ward, Rachel M. Holden, Michael A. Adams
Format: Article
Language:English
Published: Wiley 2020-11-01
Series:Physiological Reports
Subjects:
CKD
diurnal
FGF‐23
phosphate
vascular calcification
Online Access:https://doi.org/10.14814/phy2.14626

Internet

https://doi.org/10.14814/phy2.14626

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