Toll-like receptor 3-mediated inflammation by p38 is enhanced by endothelial nitric oxide synthase knockdown

Toll-like receptor 3-mediated inflammation by p38 is enhanced by endothelial nitric oxide synthase knockdown

Abstract Background Vascular dysfunction is commonly seen during severe viral infections. Endothelial nitric oxide synthase (eNOS), has been postulated to play an important role in regulating vascular homeostasis as well as propagation of the inflammatory reaction. We hypothesized that the loss of e...

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Bibliographic Details
Main Authors: Stephen R. Koch, Hyehun Choi, Eric H. Mace, Ryan J. Stark
Format: Article
Language:English
Published: BMC 2019-04-01
Series:Cell Communication and Signaling
Subjects:
eNOS
TLR3
Inflammation
Endothelial dysfunction
p38
Online Access:http://link.springer.com/article/10.1186/s12964-019-0345-3

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http://link.springer.com/article/10.1186/s12964-019-0345-3

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