Deletion of IL-33R (ST2) abrogates resistance to EAE in BALB/C mice by enhancing polarization of APC to inflammatory phenotype.

Deletion of IL-33R (ST2) abrogates resistance to EAE in BALB/C mice by enhancing polarization of APC to inflammatory phenotype.

The administration of interleukin 33 and deletion of IL-33 receptor, ST2 molecule, affects the induction of autoimmunity in different experimental models of human autoimmune diseases. The aim of this study was to analyze the effect of ST2 deletion on the induction of experimental autoimmune encephal...

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Bibliographic Details
Main Authors: Marija Milovanovic, Vladislav Volarevic, Biljana Ljujic, Gordana Radosavljevic, Ivan Jovanovic, Nebojsa Arsenijevic, Miodrag L Lukic
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2012-01-01
Series:PLoS ONE
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/23028861/?tool=EBI

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https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/23028861/?tool=EBI

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