TLR4 participates in sympathetic hyperactivity Post-MI in the PVN by regulating NF-κB pathway and ROS production

TLR4 participates in sympathetic hyperactivity Post-MI in the PVN by regulating NF-κB pathway and ROS production

Sympathetic nerve hyperactivity is a primary reason for fatal ventricular arrhythmias (VAs) following myocardial infarction (MI). Pro-inflammatory cytokines produced in the paraventricular nucleus (PVN) post-MI are associated with sympathetic overexcitation; however, the precise mechanism needs furt...

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Bibliographic Details
Main Authors: Yu Wang, Hesheng Hu, Jie Yin, Yugen Shi, Jiayu Tan, Lu Zheng, Cailing Wang, Xiaolu Li, Mei Xue, Ju Liu, Ye Wang, Yan Li, Xinran Li, Fuhong Liu, Qiang Liu, Suhua Yan
Format: Article
Language:English
Published: Elsevier 2019-06-01
Series:Redox Biology
Online Access:http://www.sciencedirect.com/science/article/pii/S2213231719300187

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http://www.sciencedirect.com/science/article/pii/S2213231719300187

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