Helicobacter pylori usurps cell polarity to turn the cell surface into a replicative niche.
Helicobacter pylori (Hp) intimately interacts with the gastric epithelial surface and translocates the virulence factor CagA into host cells in a contact-dependent manner. To study how Hp benefits from interacting with the cell surface, we developed live-cell microscopy methods to follow the fate of...
Main Authors: | , , |
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Format: | Article |
Language: | English |
Published: |
Public Library of Science (PLoS)
2009-05-01
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Series: | PLoS Pathogens |
Online Access: | http://europepmc.org/articles/PMC2669173?pdf=render |