The loss of β adrenergic receptor mediated release potentiation in a mouse model of fragile X syndrome

In fragile X syndrome, the absence of Fragile X Mental Retardation Protein (FMRP) is known to alter postsynaptic function, although alterations in presynaptic function also occur. We found that the potentiation of glutamate release induced by the β adrenergic receptor (βAR) agonist isoproterenol is...

Full description

Bibliographic Details
Main Authors: Nuria García-Font, Ricardo Martín, Magdalena Torres, María Jesus Oset-Gasque, José Sánchez-Prieto
Format: Article
Language:English
Published: Elsevier 2019-10-01
Series:Neurobiology of Disease
Subjects:
Online Access:http://www.sciencedirect.com/science/article/pii/S0969996119301330