Triamcinolone suppresses retinal vascular pathology via a potent interruption of proinflammatory signal-regulated activation of VEGF during a relative hypoxia

Triamcinolone suppresses retinal vascular pathology via a potent interruption of proinflammatory signal-regulated activation of VEGF during a relative hypoxia

We examined the effect of triamcinolone acetonide (TA), a corticosteroid, on the relationship between vascular pathophysiology and vascular endothelial growth factor (VEGF) activation in the retina of a rat model of oxygen-induced retinopathy (OIR). OIR was induced by exposure of hyperoxia (80% oxyg...

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Bibliographic Details
Main Authors: Y.H. Kim, I.Y. Chung, M.Y. Choi, Y.S. Kim, J.H. Lee, C.H. Park, S.S. Kang, G.S. Roh, W.S. Choi, J.M. Yoo, G.J. Cho
Format: Article
Language:English
Published: Elsevier 2007-06-01
Series:Neurobiology of Disease
Subjects:
TA
VEGF
OIR
Retina
Hypoxia–response proinflammatory genes
Hypoxia
Online Access:http://www.sciencedirect.com/science/article/pii/S0969996107000435

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http://www.sciencedirect.com/science/article/pii/S0969996107000435

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