iNOS is not responsible for RyR1 S-nitrosylation in mdx mice with truncated dystrophin

iNOS is not responsible for RyR1 S-nitrosylation in mdx mice with truncated dystrophin

Abstract Background Previous research indicated that nitric oxide synthase (NOS) is the key molecule for S-nitrosylation of ryanodine receptor 1 (RyR1) in DMD model mice (mdx mice) and that both neuronal NOS (nNOS) and inducible NOS (iNOS) might contribute to the reaction because nNOS is mislocalize...

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Bibliographic Details
Main Authors: Ken’ichiro Nogami, Yusuke Maruyama, Ahmed Elhussieny, Fusako Sakai-Takemura, Jun Tanihata, Jun-ichi Kira, Yuko Miyagoe-Suzuki, Shin’ichi Takeda
Format: Article
Language:English
Published: BMC 2020-07-01
Series:BMC Musculoskeletal Disorders
Subjects:
iNOS
nNOS
Duchenne muscular dystrophy
Becker muscular dystrophy
Ryanodine receptor 1 (RyR1)
Online Access:http://link.springer.com/article/10.1186/s12891-020-03501-0

Internet

http://link.springer.com/article/10.1186/s12891-020-03501-0

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