Extended role of necrotic cell death after hypoxia–ischemia-induced neurodegeneration in the neonatal rat
The relative contribution of apoptosis and necrosis after neonatal hypoxia–ischemia (HI) is still a matter of debate. Here we determined the time course of necrotic cell death after neonatal HI and its relationship to caspase-3 activation and apoptotic cell death. Necrosis was evaluated by intracere...
Main Authors: | Silvia Carloni, Andrea Carnevali, Mauro Cimino, Walter Balduini |
---|---|
Format: | Article |
Language: | English |
Published: |
Elsevier
2007-09-01
|
Series: | Neurobiology of Disease |
Subjects: | |
Online Access: | http://www.sciencedirect.com/science/article/pii/S0969996107001155 |
Similar Items
-
Protective role of autophagy in neonatal hypoxia–ischemia induced brain injury
by: Silvia Carloni, et al.
Published: (2008-12-01) -
Simvastatin reduces caspase-3 activation and inflammatory markers induced by hypoxia–ischemia in the newborn rat
by: Silvia Carloni, et al.
Published: (2006-01-01) -
Comparison of Calpain and Caspase Activities in the Adult Rat Brain after Transient Forebrain Ischemia
by: Chen Zhang, et al.
Published: (2002-08-01) -
Lysosomal rupture, necroapoptotic interactions and potential crosstalk between cysteine proteases in neurons shortly after focal ischemia
by: Münire Kilinc, et al.
Published: (2010-10-01) -
Cell Death in the Developing Brain after Hypoxia-Ischemia
by: Claire Thornton, et al.
Published: (2017-08-01)