JNK Suppresses Tumor Formation via a Gene-Expression Program Mediated by ATF2

JNK Suppresses Tumor Formation via a Gene-Expression Program Mediated by ATF2

JNK and p38 phosphorylate a diverse set of substrates and, consequently, can act in a context-dependent manner to either promote or inhibit tumor growth. Elucidating the functions of specific substrates of JNK and p38 is therefore critical for our understanding of these kinases in cancer. ATF2 is a ...

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Bibliographic Details
Main Authors: Malgorzata Gozdecka, Stephen Lyons, Saki Kondo, Janet Taylor, Yaoyong Li, Jacek Walczynski, Gerald Thiel, Wolfgang Breitwieser, Nic Jones
Format: Article
Language:English
Published: Elsevier 2014-11-01
Series:Cell Reports
Online Access:http://www.sciencedirect.com/science/article/pii/S2211124714009127

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http://www.sciencedirect.com/science/article/pii/S2211124714009127

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