Emodin Inhibits ATP-Induced Proliferation and Migration by Suppressing P2Y Receptors in Human Lung Adenocarcinoma Cells

Emodin Inhibits ATP-Induced Proliferation and Migration by Suppressing P2Y Receptors in Human Lung Adenocarcinoma Cells

Background/Aims: Extracellular ATP performs multiple important functions via activation of P2 receptors on the cell surface. P2Y receptors play critical roles in ATP evoked response in human lung adenocarcinoma cells (A549 cells). Emodin is an anthraquinone derivative originally isolated from Chines...

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Bibliographic Details
Main Authors: Xia Wang, Long Li, Ruijuan Guan, Danian Zhu, Nana Song, Linlin Shen
Format: Article
Language:English
Published: Cell Physiol Biochem Press GmbH & Co KG 2017-11-01
Series:Cellular Physiology and Biochemistry
Subjects:
Emodin
ATP
A549cells
Suramin
NF-κB
Online Access:https://www.karger.com/Article/FullText/485495

Internet

https://www.karger.com/Article/FullText/485495

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