Neutralization of Lipocalin-2 Diminishes Stroke-Reperfusion Injury

Neutralization of Lipocalin-2 Diminishes Stroke-Reperfusion Injury

Oxidative stress is a key contributor to the pathogenesis of stroke-reperfusion injury. Neuroinflammatory peptides released after ischemic stroke mediate reperfusion injury. Previous studies, including ours, have shown that lipocalin-2 (LCN2) is secreted in response to cerebral ischemia to promote r...

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Bibliographic Details
Main Authors: Guona Wang, Yi-Chinn Weng, I-Chen Chiang, Yu-Ting Huang, Yi-Chu Liao, Yi-Chun Chen, Cheng-Yuan Kao, Yu-Li Liu, Tsong-Hai Lee, Wen-Hai Chou
Format: Article
Language:English
Published: MDPI AG 2020-08-01
Series:International Journal of Molecular Sciences
Subjects:
Lipocalin-2
stroke
reperfusion injury
oxidative stress
immunotherapy
neutrophils
Online Access:https://www.mdpi.com/1422-0067/21/17/6253

Internet

https://www.mdpi.com/1422-0067/21/17/6253

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