NADPH oxidase 2 does not contribute to early reperfusion-associated reactive oxygen species generation following transient focal cerebral ischemia

NADPH oxidase 2 does not contribute to early reperfusion-associated reactive oxygen species generation following transient focal cerebral ischemia

Excess production of reactive oxygen species (ROS) critically contributes to occurrence of reperfusion injury, the paradoxical response of ischemic brain tissue to restoration of cerebral blood flow. However, the enzymatic sources of ROS generation remain to be unclear. This study examined Nox2-cont...

Full description

Bibliographic Details
Main Authors: Yuan Zhang, Ting Wang, Ke Yang, Ji Xu, Jian-ming Wu, Wen-lan Liu
Format: Article
Language:English
Published: Wolters Kluwer Medknow Publications 2016-01-01
Series:Neural Regeneration Research
Subjects:
nerve regeneration; NADPH oxidase; cerebral ischemia; Nox family; reactive oxygen species; reperfusion; central nervous system; stroke; blood flow; neural regeneration
Online Access:http://www.nrronline.org/article.asp?issn=1673-5374;year=2016;volume=11;issue=11;spage=1773;epage=1778;aulast=Zhang

Internet

http://www.nrronline.org/article.asp?issn=1673-5374;year=2016;volume=11;issue=11;spage=1773;epage=1778;aulast=Zhang

Similar Items