MicroRNA-323a-3p Promotes Pressure Overload-Induced Cardiac Fibrosis by Targeting TIMP3

MicroRNA-323a-3p Promotes Pressure Overload-Induced Cardiac Fibrosis by Targeting TIMP3

Background/Aims: Cardiac fibrosis is a major cause of diverse cardiovascular diseases. MicroRNAs have recently been proven a novel class of regulators of cardiac fibrosis. In this study, we sought to investigate the role of miR-323a-3p and its mechanisms in regulating cardiac fibrosis. Methods: The...

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Bibliographic Details
Main Authors: JingChao  Zhang, Yan Lang, LongHui Guo, Yu Pei, Shuang Hao, ZhenXing Liang, Gang Su, LiLiang Shu, Hai Liu, Chen Huang, Jing Xu
Format: Article
Language:English
Published: Cell Physiol Biochem Press GmbH & Co KG 2018-11-01
Series:Cellular Physiology and Biochemistry
Subjects:
miR-323-3p
TAC
Cardiac fibrosis
TIMP3
TGFβ
Online Access:https://www.karger.com/Article/FullText/495059

Internet

https://www.karger.com/Article/FullText/495059

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